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In ME/CFS and Long COVID, research suggests that the mitochondria are damaged by chronic oxidative stress and fail to produce ATP efficiently

Sci Rep 15:3248

However, our study did not directly assess mitochondrial function or ROS levels, and thus, this mechanism remains speculative

172,173 In support of these findings, in vivo studies using mouse stroke models have demonstrated that ATX knockout reduces LPA levels, improves vascular permeability, enhances cerebral blood flow, and decreases infarct volume, collectively reducing HCM-associated vascular dysfunction