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The variation in C max within the normal renal function group was larger than the renal impairment groups owing to outlying profiles with low exposure

In addition to mitochondrial-targeted therapy, mitochondrial gene therapy is emerging as a novel research domain

It is hypothesized that HOXA10 could drive the misplacing of endometrial cells during embryonic life and causing endometriosis, but there is currently no in vivo proof that HOXA10 dysregulation during uterine development may be a possible contributor to the etiology of endometriosis later in life [79, 80]