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doi: 10.1186/1743-7075-9-35

Obesity and ageing: two sides of the same coin

Social interaction deficits parallel evidence that prenatal cannabinoid exposure disrupts sociability via altered endocannabinoid and oxytocin signaling [54, 55, 56], while broader affective and motivational abnormalities align with persistent synaptic dysregulation in corticolimbic circuits following early-life adversity [57, 58, 59]

Given that synapses degrade and protective mechanisms fade as a function of normal ageing, it might be expected that synaptic dysfunction and loss is more pronounced in LLD [242, 243]

Subtypes A1 and A3 bind to G i -coupled GPCRs thereby inactivating adenylate cyclase and decreasing intracellular cAMP levels