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The expression of genes involved in gluconeogenesis, glycogenolysis, lipid synthesis and transport, and catabolism were altered in untreated Hz animals and the expression of most of them was normalized by IGF-1 therapy

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For example, a landmark study on rat Achilles tendon healing found that BPC-157 administration led to a functional, structural, and biomechanical recovery that was almost indistinguishable from a healthy, uninjured tendon

However, when ROS production exceeds the capacity of antioxidant defense systems, oxidative stress ensues, resulting in damage to lipids, proteins, and nucleic acids, and triggering cellular dysfunction, senescence, or death [11]