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Thus, the transfer of apoC-I from HDL to TG-rich lipoproteins facilitate atherogenic remnant formation, suggesting a dual role of apoC-I: (1) preventing remnant formation and premature atherosclerosis if attached to HDL and (2) promoting remnant formation and atherosclerosis if transferred to TG-rich lipoproteins [118]

People Are Also Reading This Medically reviewed by Mia Dacumos, MD Nephrology Makati Medical Center

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