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MEFs lacking Bmal1 showed lower CI respiration, reduced SAM levels and decreased NDUFS2 methylation (Fig

In particular, mitochondrial dysfunction is a key finding in reactive oxygen species (ROS)-induced PD pathology [4,5,6,7]

Modeling of TREX1-dependent autoimmune disease using human stem cells highlights L1 accumulation as a source of neuroinflammation

Gamma-glutamyl transpeptidase is located in the cell membrane and serves to shuttle glutathione to the cell surface to react with an amino acid

The model was first fitted to the training concentrations (37.5 and 25 M doxorubicin), and then used to simulate the test concentrations (50, 12.5 and 10 M doxorubicin) as a means of blind validation, showing good accordance between the experimental and in silico outputs for all concentrations of doxorubicin