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They are two different classes of molecule with two different mechanisms and two different evidence trails, one of which happens to include a body of human physiological data the other simply doesnt have, owing entirely to the fact that one of them is something the body already makes

Front Pharmacol 15:1405423

An examination using murine models of coronary artery tying appeared to exhibit results which implied that TB-500 might elevate integrin-associated kinase (ILK) and protein kinase B operations in the heart, possibly boosting early cardiomyocyte endurance and seemingly enhancing heart performance.(15)The experts also suggested that TB-500 might support the movement of myocardial and endothelial cells in the fetal heart and maintains this capability in mature cardiomyocytes

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